Pathology Graduate Training Program
Cellular and Molecular Pathology (CMP) -
Current Graduate Students

Name: Ricardo Londono
Email: londono.ricardo@medstudent.pitt.edu
Lab Phone: 412-624-5252
Mentor Name: Stephen Badylak, MD, PhD

Education:
Name of school attended: Florida International University
Undergraduate degree: B.S. Electrical and Computer Engineering

Research Project Information:

My research is focused upon the properties and biologic activities of matricryptic peptides generated from scaffolds prepared from mammalian extracellular matrix (ECM) during the early events of tissue reconstruction and remodeling.

Publications:

VanEpps JS, Londono R, Nieponice A, Vorp DA. Design and validation of a system to simulate coronary flexure dynamics on arterial segments perfused ex vivo. Biomech Model Mechanobiol. 2009 Feb;8(1):57-66. Epub 2008 Feb 23.

Tottey S, Corselli M, Jeffries EM, Londono R, Peault B, Badylak SF. Extracellular matrix degradation products and low oxygen conditions enhance the regenerative potential of perivascular stem cells. Tissue Eng Part A. 2010 Jul 23.

Abstracts/Presentations

  1. Londono, R; VanEpps, JS; Chew WD; Vorp, DA. Biological Assessment of a Novel Cyclic Bending Device for Ex-vivo Vascular Organ Culture. SACNAS. Denver, CO, 2005
  2. Ricardo Londono; J. Scot VanEpps; Robert J. Toth; Alejandro Nieponice; David A. Vorp. Development of a Novel Device to Apply Coronary-Like Bending Motion to Intact Vascular Segments Perfused Ex-vivo. Pittsburgh Tissue Engineering Initiative, Pittsburgh, PA, 2006
  3. Ricardo Londono; Stephen Tottey; Janet Reign; Stephen Badylak. Oxygen Dependant Mitogenic and Chemotactic Properties of Urinary Bladder Matrix and Small Intestinal Submucosa on MG71 Cells. University of Pittsburgh MSTP Annual Retreat 2008
  4. Ricardo Londono; Stephen Tottey; Stephen Badylak. Chemotactic and Antimicrobial Properties of Macrophage- and Enzyme-Digested Extracellular Matrix. University of Pittsburgh MSTP Annual Retreat 2009
  5. Ricardo Londono; Stephen Tottey; Stephen Badylak. Monocyte phenotype modulation with different levels of biologic scaffold decellularization